Publication	Data		Fish	Anatomy
Pei et al., 2018	text only		lef1la021271Tg/la021271Tg	neuromast development disrupted, abnormal post-vent region lacks all parts of type posterior lateral line neuromast, abnormal
Xie et al., 2017	Fig. 1		lef1zd11/zd11; w25Tg	caudal periventricular hypothalamus GABAergic neuron decreased amount, abnormal caudal periventricular hypothalamus GFP expression decreased amount, abnormal caudal periventricular hypothalamus GFP expression decreased distribution, abnormal caudal periventricular hypothalamus serotonin secreting cell decreased amount, abnormal
	Fig. 2		lef1zd11/zd11	brain crhbp expression decreased amount, abnormal brain dkk1b expression decreased amount, abnormal brain grin2cb expression decreased amount, abnormal brain htr1ab expression decreased amount, abnormal brain lef1 expression decreased amount, abnormal brain penka expression decreased amount, abnormal brain si:dkey-219c10.4 expression decreased amount, abnormal brain slc6a4b expression decreased amount, abnormal brain sp5a expression decreased amount, abnormal brain sp5l expression decreased amount, abnormal central nervous system notum1a expression decreased amount, abnormal
	Fig. 3		lef1zd11/zd11	whole organism decreased length, abnormal
Matsuda et al., 2013	Fig. 1		zf106Tg + MO3-lef1 + MO4-tp53	neuromast decreased distance neuromast, abnormal neuromast deposition process quality, abnormal
	Fig. 2		zf106Tg + MO3-lef1 + MO4-tp53	posterior lateral line primordium cell division decreased process quality, abnormal posterior lateral line primordium cell population proliferation decreased process quality, abnormal
	Fig. 3		zf106Tg + MO3-lef1 + MO4-tp53	neuromast deposition process quality, abnormal posterior lateral line neuromast primordium migration process quality, abnormal
Wada et al., 2013	Fig. 2		WT + MO3-lef1	neuromast has fewer parts of type neuromast hair cell, abnormal
Ota et al., 2012	Fig. 2		WT + MO2-lef1 ,   rw0130aTg + MO2-lef1 ,   rw0130aTg + MO2-lef1 + MO4-tp53	cell proliferation in midbrain decreased occurrence, abnormal midbrain development disrupted, abnormal optic tectum decreased size, abnormal
Wang et al., 2012	Fig. 2		lef1zd11/zd11 (AB)	caudal fin aplastic, abnormal fin decreased size, abnormal hypothalamus posterior region decreased size, abnormal
McGraw et al., 2011	Fig. 1		lef1nl2/nl2; zf106Tg ,   lef1nl2/nl2 (WIK/AB)	posterior lateral line neuromast posterior-most region absent, abnormal posterior lateral line primordium increased length, abnormal
	Fig. 2		lef1nl2/nl2; zf106Tg ,   zf106Tg + MO4-lef1	posterior lateral line neuromast mislocalised anteriorly, abnormal posterior lateral line truncated, abnormal
	Fig. 4		lef1nl2/nl2 (WIK/AB)	posterior lateral line truncated, abnormal
	Fig. S1		lef1nl2/nl2; nl1Tg	posterior lateral line nerve truncated, abnormal posterior lateral line neuromast posterior-most region absent, abnormal
	Fig. S2		lef1nl2/nl2 (WIK/AB)	caudal fin absent, abnormal caudal fin lepidotrichium hypoplastic, abnormal ceratobranchial 5 tooth decreased amount, abnormal pectoral fin lepidotrichium hypoplastic, abnormal
	Fig. S3		lef1nl2/nl2; nl1Tg	posterior lateral line truncated, abnormal
Valdivia et al., 2011	Fig. 1		ia4Tg + MO3-lef1 ,   lef1u767/u767 ,   WT + MO3-lef1	posterior lateral line development decreased process quality, abnormal posterior lateral line neuromast decreased amount, abnormal Wnt signaling pathway decreased process quality, abnormal
	Fig. 2		lef1u767/u767	posterior lateral line development decreased process quality, abnormal posterior lateral line neuromast decreased amount, abnormal
	Fig. 4		lef1u767/u767; zf106Tg ,   zf106Tg + MO3-lef1	posterior lateral line development decreased process quality, abnormal posterior lateral line neuromast decreased amount, abnormal
	Fig. 5		lef1u767/u767; zf106Tg	posterior lateral line primordium self-renewal, abnormal regulation of cell proliferation involved in tissue homeostasis decreased process quality, abnormal
	Fig. S1		lef1u767/u767	anal fin decreased length, abnormal caudal fin decreased length, abnormal dorsal fin decreased length, abnormal fin development decreased process quality, abnormal pectoral fin absent, abnormal
Tee et al., 2009	Fig. 7		WT + MO1-lef1	somite decreased amount, abnormal
Russek-Blum et al., 2008	Fig. 8		WT + MO2-lef1	diencephalon dopaminergic neuron increased amount, abnormal
	Fig. S6		WT + MO2-lef1	diencephalon dopaminergic neuron increased amount, abnormal
Nyholm et al., 2007	Fig. 6		WT + MO2-lef1	brain wholly posteriorized, abnormal midbrain morphology, abnormal
Lee et al., 2006	Fig. 4		WT + MO2-lef1	forebrain neuron development disrupted, abnormal hypothalamus neuron morphology, abnormal
	Fig. 5		WT + MO2-lef1	hypothalamus axon decreased amount, abnormal hypothalamus cell differentiation disrupted, abnormal hypothalamus neuron decreased amount, abnormal
Ishitani et al., 2005	Fig. 1		WT + MO1-lef1 ,   WT + MO2-lef1	cartilage morphogenesis disrupted, abnormal caudal fin morphology, abnormal cranial cartilage morphology, abnormal dorsal root ganglion decreased amount, abnormal dorsal root ganglion mislocalised, abnormal mandibular arch skeleton decreased size, abnormal neural crest cell development disrupted, abnormal pigmentation disrupted, abnormal post-anal tail morphogenesis disrupted, abnormal whole organism decreased pigmentation, abnormal whole organism decreased size, abnormal
	Fig. 2		WT + MO1-lef1	neural crest cell migration disrupted, abnormal trunk neural crest cell positional polarity, abnormal
	Fig. 3		WT + MO1-lef1 ,   w25Tg + MO1-lef1	melanocyte decreased amount, abnormal pigment cell differentiation disrupted, abnormal trunk neural crest cell decreased amount, abnormal
Dorsky et al., 2002	Fig. 2		WT + MO1-lef1	paraxial mesoderm decreased size, abnormal post-vent region decreased length, abnormal post-vent region truncated, abnormal