FIGURE

Fig. 1

ID

ZDB-FIG-240313-15

Publication

Auman et al., 2023 - Zebrafish smarcc1a mutants reveal requirements for BAF chromatin remodeling complexes in distinguishing the atrioventricular canal from the cardiac chambers

Other Figures

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Fig. 1

The sk29 mutation causes multiple embryonic defects. (A, B) Lateral views of live embryos at 48 hpf, anterior to the left, dorsal to the top. In comparison to wild-type embryos (wt), sk29 mutant embryos (mut) have a slightly shorter body axis, decreased pigmentation, pericardial edema, and a thickened yolk extension. (C, D) Lateral views at 48 hpf, anterior to the top, dorsal to the left. In sk29 mutants, cardiac chambers (arrowheads) fail to expand and remain relatively linear, with little apparent morphological distinction of the AVC. (E, F) Dorsal views at 72 hpf, anterior to the top. Pectoral fins (arrows) are absent (asterisks) in sk29 mutants. (G, H) Lateral views at 48 hpf, anterior to the top, dorsal to the left. Retinal pigmentation is abnormal in sk29 mutants.

Expression Data

Expression Detail

Antibody Labeling

Phenotype Data

Fish:	smarcc1a^sk29/sk29
Observed In:	atrioventricular canal extension heart contraction melanocyte pectoral fin pericardium retina retinal pigmented epithelium whole organism
Stage Range:	Prim-15 to Protruding-mouth

Phenotype Detail

Acknowledgments

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