Publication	Data		Fish	Anatomy
Yang et al., 2021	Fig. 5		AB + MO1-shha	pancreas primordium ab2-isl labeling decreased amount, abnormal pancreas primordium Ab8-ins labeling decreased amount, abnormal
Hong et al., 2020	Fig. 1		shhahg126/hg126	caudal fin curled, abnormal eye fused with eye, abnormal somite U-shaped, abnormal
Nittoli et al., 2019	Fig. 7		shhatbx392/tbx392	somite ventral side uncx expression spatial pattern, abnormal
Rurale et al., 2019	Fig. 5		AB + MO1-shha	brain glis3 expression absent, abnormal endoderm glis3 expression absent, abnormal
Sugimoto et al., 2017	Fig. 3 S2		shhavcc8Gt/+; vcc9Tg/+ (EKW) ,   shhavcc8Gt/vcc8Gt; vcc9Tg/+ (EKW)	pectoral fin decreased size, abnormal whole organism shha expression absent, abnormal whole organism shha expression decreased amount, abnormal
Zhang et al., 2017	Fig. 2		AB + MO1-shha	eye pax6a expression decreased amount, abnormal forebrain pax6a expression decreased amount, abnormal hindbrain pax6a expression decreased amount, abnormal
	Fig. 5		AB + MO1-shha	midbrain hindbrain boundary morphology, abnormal
Osborn et al., 2011	Fig. 1		shhatbx392/tbx392	segmental plate anterior region has fewer parts of type adaxial cell, abnormal skeletal muscle tissue development disrupted, abnormal
	Fig. 2		shhatbx392/tbx392	skeletal muscle tissue development delayed, abnormal
	Fig. 3		shhatbx392/tbx392	adaxial cell decreased amount, abnormal
Jackman et al., 2010	Fig. 3		WT + MO1-shha	odontogenesis arrested, abnormal whole organism lacks all parts of type ceratobranchial 5 tooth, abnormal
Teoh et al., 2010	Fig. 5		shhaunspecified/unspecified	swim bladder physical object quality, abnormal
Philipp et al., 2008	Fig. 6		WT + MO1-shha	slow muscle cell nucleus decreased amount, abnormal somite muscle pioneer decreased amount, abnormal
Stenkamp et al., 2008	Fig. 2		shhatbx392/+	retinal cone cell decreased amount, abnormal retinal cone cell irregular spatial pattern, abnormal
Feng et al., 2006	Fig. 2		shhatbx392/tbx392 (AB)	muscle cell differentiation decreased occurrence, abnormal somite has extra parts of type muscle precursor cell, abnormal
Phenotype Annotation (1994-2006)	Fig. for (tbq70)		shhatbq70	anatomical system quality, abnormal eye decreased size, abnormal fin decreased size, abnormal heart quality, abnormal myoseptum quality, abnormal pectoral fin quality, abnormal post-vent region quality, abnormal sensory system quality, abnormal somite quality, abnormal surface structure quality, abnormal whole organism shape, abnormal
	Fig. for (tbx392)		shhatbx392/tbx392	anatomical system quality, abnormal eye decreased size, abnormal fin decreased size, abnormal heart quality, abnormal myoseptum quality, abnormal pectoral fin quality, abnormal post-vent region quality, abnormal sensory system quality, abnormal somite quality, abnormal surface structure quality, abnormal whole organism shape, abnormal
	Fig. for (tq252)		shhatq252/tq252	anatomical system quality, abnormal fin decreased size, abnormal floor plate quality, abnormal musculoskeletal movement disrupted, abnormal myoseptum quality, abnormal notochord quality, abnormal pectoral fin quality, abnormal somite quality, abnormal surface structure quality, abnormal whole organism lethal (sensu genetics), abnormal
Wallace et al., 2003	Fig. 10		shhatbx392/tbx392	liver increased size, abnormal primary islet absent, abnormal
	Fig. 9		shhatbx392/tbx392	esophageal epithelium disorganized, abnormal esophageal epithelium epithelial cell differentiation disrupted, abnormal esophagus malformed, abnormal
Coutelle et al., 2001	Fig. 1		shhatbx392/tbx392	adaxial cell physical object quality, abnormal muscle pioneer absent, abnormal skeletal muscle fiber development process quality, abnormal slow muscle cell decreased amount, abnormal
	Fig. 4		shhatbx392/tbx392	adaxial cell immature, abnormal striated muscle cell differentiation delayed, abnormal
	Fig. 6		shhatbx392/tbx392	slow muscle cell decreased amount, abnormal striated muscle cell differentiation process quality, abnormal
Grandel et al., 2000	Fig. 1		shhatq252/tq252	fin fold pectoral fin bud disorganized, abnormal pectoral fin endoskeletal disc decreased size, abnormal pectoral girdle morphology, abnormal
Neumann et al., 1999	Fig. 9		shhatq252/tq252	pectoral fin actinotrichium decreased size, abnormal pectoral fin bud decreased size, abnormal pectoral fin cleithrum decreased length, abnormal pectoral fin endoskeletal disc decreased size, abnormal pectoral fin scapulocoracoid decreased size, abnormal pectoral fin skeleton cell decreased amount, abnormal
Schauerte et al., 1998	Fig. 3		shhatq252/tq252 ,   shhatbq70/tbq70	horizontal myoseptum absent, abnormal somite decreased size, abnormal somite U-shaped, abnormal
	Fig. 6		shhatbq70/tbq70	retina development in camera-type eye disrupted, abnormal retinal ganglion cell axon guidance disrupted, abnormal
Brand et al., 1996	Table 1		shhatq252/tq252	floor plate hypoplastic, abnormal myoseptum aplastic, abnormal notochord morphology, abnormal pectoral fin decreased size, abnormal whole organism movement behavioral quality, abnormal whole organism viability, abnormal
Chen et al., 1996	Table 1		shhatq252/tq252	blood circulation arrested, abnormal blood circulation delayed, abnormal
Granato et al., 1996	Table 4		shhatbx392/tbx392 ,   shhatq252/tq252 ,   shhatbq70/tbq70	larval locomotory behavior process quality, abnormal whole organism decreased behavioural activity, abnormal
van Eeden et al., 1996	Fig. 3		shhatq252/tq252	pectoral fin decreased length, abnormal pectoral fin variability of size, abnormal
	Table 1		shhatq252/tq252	horizontal myoseptum absent, abnormal neural tube decreased thickness, abnormal pectoral fin decreased size, abnormal
	Table 2		shhatq252/tq252	pectoral fin decreased size, abnormal
van Eeden et al., 1996	Table 3		shhatq252/tq252	blood circulation disrupted, abnormal horizontal myoseptum hypoplastic, abnormal pectoral fin decreased size, abnormal whole organism movement behavioral quality, abnormal