Phenotype
|
Fish
|
Conditions
|
Figures
|
retina cell decreased amount, abnormal
|
WT + MO1-id2a
|
control
|
Fig. 1
from Uribe et al., 2012
|
retina rspo1 expression increased amount, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 7
from Uribe et al., 2012
|
retina tcf7l1b expression increased amount, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 7
from Uribe et al., 2012
|
liver hhex expression decreased amount, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. S13
from Xu et al., 2016
|
retina hes6 expression increased amount, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 3
from Uribe et al., 2012
|
brain decreased size, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 1
from Uribe et al., 2010
|
liver apoptotic process increased occurrence, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 4
from Khaliq et al., 2015
|
amacrine cell amount, ameliorated
|
WT + MO1-id2a
|
chemical treatment: DAPT
|
Fig. 2
from Uribe et al., 2012
|
retina notch1a expression increased amount, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 3
from Uribe et al., 2012
|
retinal cone cell amount, ameliorated
|
WT + MO1-id2a
|
chemical treatment: DAPT
|
Fig. 2
from Uribe et al., 2012
|
retina cell population proliferation process quality, ameliorated
|
WT + MO1-id2a
|
chemical treatment: DAPT
|
Fig. 1
from Uribe et al., 2012
|
retina dla expression increased distribution, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 3
from Uribe et al., 2012
|
retina her4.1 expression increased amount, abnormal
|
WT + MO1-id2a
|
control
|
Fig. 3
from Uribe et al., 2012
|
retinal inner nuclear layer cell undifferentiated, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 2
from Uribe et al., 2010
|
S phase delayed, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 5
from Uribe et al., 2010
|
retina ascl1a expression increased amount, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 3
from Uribe et al., 2012
|
retina dla expression increased amount, abnormal
|
WT + MO1-id2a
|
control
|
Fig. 3
from Uribe et al., 2012
|
Muller cell decreased amount, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 2
from Uribe et al., 2010
|
amacrine cell absent, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 2 ,
Fig. 5
from Uribe et al., 2012
Fig. 2
from Uribe et al., 2010
|
eye photoreceptor cell differentiation decreased process quality, abnormal
|
WT + MO1-id2a
|
control
|
Fig. 1
from Uribe et al., 2012
|
retina skp2 expression increased amount, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 7
from Uribe et al., 2012
|
retinal cone cell differentiation process quality, ameliorated
|
WT + MO1-id2a
|
chemical treatment: DAPT
|
Fig. 2
from Uribe et al., 2012
|
retina ascl1a expression increased distribution, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 3
from Uribe et al., 2012
|
retinal cone cell zpr-1 labeling absent, abnormal
|
WT + MO1-id2a
|
control
|
Fig. 2
from Uribe et al., 2012
|
retinal rod cell decreased amount, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 2
from Uribe et al., 2010
|
regulation of neural retina development disrupted, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 4
from Uribe et al., 2010
|
eye cell decreased amount, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 4
from Uribe et al., 2012
|
intestinal bulb decreased size, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 3
from Khaliq et al., 2015
|
retina cell amount, ameliorated
|
WT + MO1-id2a
|
chemical treatment: DAPT
|
Fig. 1
from Uribe et al., 2012
|
retina cdkn1a expression increased amount, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 7
from Uribe et al., 2012
|
retina fzd6 expression increased amount, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 7
from Uribe et al., 2012
|
retinal bipolar neuron absent, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 2
from Uribe et al., 2010
|
retinal cone cell zpr-1 labeling amount, ameliorated
|
WT + MO1-id2a
|
chemical treatment: DAPT
|
Fig. 2
from Uribe et al., 2012
|
retinal outer nuclear layer cell undifferentiated, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 2
from Uribe et al., 2010
|
camera-type eye photoreceptor cell differentiation disrupted, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 5
from Uribe et al., 2012
Fig. 2
from Uribe et al., 2010
|
embryonic retina morphogenesis in camera-type eye delayed, abnormal
|
WT + MO1-id2a
|
chemical treatment: 5-bromo-2'-deoxyuridine
|
Fig. 7
from Uribe et al., 2010
|
retina layer formation disrupted, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 1
from Uribe et al., 2010
|
retina cdk6 expression increased amount, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 7
from Uribe et al., 2012
|
retina ccnd1 expression increased amount, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 7
from Uribe et al., 2012
|
S phase delayed, abnormal
|
WT + MO1-id2a
|
chemical treatment: 5-bromo-2'-deoxyuridine
|
Fig. 7
from Uribe et al., 2010
|
liver decreased size, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 3
from Khaliq et al., 2015
|
amacrine cell ab-5e11 labeling amount, ameliorated
|
WT + MO1-id2a
|
chemical treatment: DAPT
|
Fig. 2
from Uribe et al., 2012
|
retinal cone cell absent, abnormal
|
WT + MO1-id2a
|
control
|
Fig. 2 ,
Fig. 5
from Uribe et al., 2012
Fig. 2
from Uribe et al., 2010
|
retinal bipolar neuron differentiation disrupted, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 2
from Uribe et al., 2010
|
amacrine cell differentiation process quality, ameliorated
|
WT + MO1-id2a
|
chemical treatment: DAPT
|
Fig. 2
from Uribe et al., 2012
|
embryonic retina morphogenesis in camera-type eye delayed, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 4 ,
Fig. 5
from Uribe et al., 2010
|
retina cell population proliferation process quality, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 1 ,
Fig. 4
from Uribe et al., 2012
|
retinal outer nuclear layer unstructured, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 1
from Uribe et al., 2010
|
retina dlc expression increased amount, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 3
from Uribe et al., 2012
|
eye decreased size, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 4
from Uribe et al., 2012
Fig. 1 ,
Fig. 5
from Uribe et al., 2010
|
retinal cone cell differentiation arrested, abnormal
|
WT + MO1-id2a
|
control
|
Fig. 2
from Uribe et al., 2012
|
amacrine cell ab-5e11 labeling absent, abnormal
|
WT + MO1-id2a
|
control
|
Fig. 2
from Uribe et al., 2012
|
retina notch1a expression increased distribution, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 3
from Uribe et al., 2012
|
amacrine cell differentiation arrested, abnormal
|
WT + MO1-id2a
|
control
|
Fig. 2
from Uribe et al., 2012
|
retinal inner nuclear layer unstructured, abnormal
|
WT + MO1-id2a
|
standard conditions
|
Fig. 1
from Uribe et al., 2010
|
liver DsRed expression decreased amount, abnormal
|
gz15Tg + MO1-id2a
|
standard conditions
|
Fig. S1
from Khaliq et al., 2015
|
liver decreased size, abnormal
|
gz15Tg + MO1-id2a
|
standard conditions
|
Fig. S1
from Khaliq et al., 2015
|
liver DsRed expression decreased amount, abnormal
|
gz15Tg + MO1-id2a + MO4-tp53
|
standard conditions
|
Fig. S1
from Khaliq et al., 2015
|
liver decreased size, abnormal
|
gz15Tg + MO1-id2a + MO4-tp53
|
standard conditions
|
Fig. S1
from Khaliq et al., 2015
|
liver cell population proliferation decreased occurrence, abnormal
|
s870Tg + MO1-id2a
|
standard conditions
|
Fig. 4
from Khaliq et al., 2015
|
intrahepatic bile duct decreased branchiness, abnormal
|
gz15Tg; um14Tg + MO1-id2a
|
standard conditions
|
Fig. 3
from Khaliq et al., 2015
|
liver decreased size, abnormal
|
gz15Tg; um14Tg + MO1-id2a
|
standard conditions
|
Fig. 3
from Khaliq et al., 2015
|
intrahepatic bile duct epithelial cell GFP expression decreased amount, abnormal
|
gz15Tg; um14Tg + MO1-id2a
|
standard conditions
|
Fig. 3
from Khaliq et al., 2015
|
intrahepatic bile duct aggregated, abnormal
|
gz15Tg; um14Tg + MO1-id2a
|
standard conditions
|
Fig. 3
from Khaliq et al., 2015
|
intrahepatic bile duct epithelial cell GFP expression spatial pattern, abnormal
|
gz15Tg; um14Tg + MO1-id2a
|
standard conditions
|
Fig. 3
from Khaliq et al., 2015
|
liver EGFP expression absent, abnormal
|
jh1Tg; um14Tg + MO1-id2a
|
standard conditions
|
Fig. 3
from Khaliq et al., 2015
|
intestine pdx1 expression decreased distribution, abnormal
|
WT + MO1-fhl1b + MO1-id2a + MO2-fhl1b
|
standard conditions
|
Fig. S13
from Xu et al., 2016
|
intestine hhex expression decreased distribution, abnormal
|
WT + MO1-fhl1b + MO1-id2a + MO2-fhl1b
|
standard conditions
|
Fig. S13
from Xu et al., 2016
|
posterior pancreatic bud hhex expression increased distribution, abnormal
|
WT + MO1-fhl1b + MO1-id2a + MO2-fhl1b
|
standard conditions
|
Fig. S13
from Xu et al., 2016
|
posterior pancreatic bud pdx1 expression increased distribution, abnormal
|
WT + MO1-fhl1b + MO1-id2a + MO2-fhl1b
|
standard conditions
|
Fig. S13
from Xu et al., 2016
|
eye cell amount, ameliorated
|
WT + MO1-id2a + MO1-notch1a
|
standard conditions
|
Fig. 4
from Uribe et al., 2012
|
amacrine cell amount, ameliorated
|
WT + MO1-id2a + MO1-notch1a
|
control
|
Fig. 5
from Uribe et al., 2012
|
camera-type eye photoreceptor cell differentiation process quality, ameliorated
|
WT + MO1-id2a + MO1-notch1a
|
control
|
Fig. 5
from Uribe et al., 2012
|
retinal cone cell amount, ameliorated
|
WT + MO1-id2a + MO1-notch1a
|
control
|
Fig. 5
from Uribe et al., 2012
|
retina cell population proliferation process quality, ameliorated
|
WT + MO1-id2a + MO1-notch1a
|
standard conditions
|
Fig. 4
from Uribe et al., 2012
|