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ZDB-FIG-110624-47

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Putiri et al., 2011 - The zebrafish maternal-effect gene mission impossible encodes the DEAH-box helicase Dhx16 and is essential for the expression of downstream endodermal genes

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Fig. s1

The nuclear and cell division cycle, as well as the f-actin enrichment at the YSL, appear normal in mis mutant embryos. Wild-type (A,C,E) and mis mutant (B,D,F) embryos were fixed during epiboly (65% epiboly (A,B), 75% epiboly (C–F)) and labeled with phalloidin (green) and DAPI (blue) to detect f-actin and DNA, respectively. No obvious defects are observed with regards to the nuclear and cell division cycles, as well as in the formation of cell adhesive membranes and the f-actin network within the YSL (arrows). (A,B) are animal views and (C–F) are side views centered on the blastoderm margin. (E,F) are higher magnifications of the panels shown in (C,D), respectively, with focus on the membranes of the EVL.

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Reprinted from Developmental Biology, 353(2), Putiri, E., and Pelegri, F., The zebrafish maternal-effect gene mission impossible encodes the DEAH-box helicase Dhx16 and is essential for the expression of downstream endodermal genes, 275-289, Copyright (2011) with permission from Elsevier. Full text @ Dev. Biol.