Publication	Data		Fish	Anatomy
Tanaka et al., 2018	Fig. 2		nkhspGFF3AEt + MO1-kcnh6a	membrane repolarization during AV node cell action potential process quality, abnormal membrane repolarization during cardiac muscle cell action potential process quality, abnormal membrane repolarization during ventricular cardiac muscle cell action potential process quality, abnormal
	Fig. 3		nkhspGFF3AEt + MO1-kcnh6a	heart contraction process quality, abnormal
	Fig. 4		nkhspGFF3AEt + MO1-kcnh6a	heart contraction process quality, abnormal
Bezzerides et al., 2017	Fig. 8		kcnh6atb218/tb218	atrioventricular canal functionality, abnormal heart contraction process quality, abnormal
Benz et al., 2016	Fig. 6		kcnh6am230/m230 ,   kcnh6am230/+	cardiac muscle cell action potential involved in contraction decreased duration, abnormal
Liu et al., 2016	Fig. 7		kcnh6atb218/tb218	heart contraction decreased rate, abnormal
Jou et al., 2013	Fig. 1		WT + MO1-kcnh6a	membrane repolarization during cardiac muscle cell action potential decreased process quality, abnormal ventricular cardiac muscle cell action potential absent, abnormal ventricular cardiac muscle cell action potential increased duration, abnormal
Meder et al., 2011	Fig. 1		kcnh6atb218/tb218	cardiac ventricle functionality, abnormal heart contraction decreased rate, abnormal regulation of ventricular cardiac muscle cell membrane repolarization process quality, abnormal
Chopra et al., 2010	Fig. 2		WT + MO2-kcnh6a	blood circulation disrupted, abnormal cardiac conduction arrested, abnormal cardiac conduction decreased rate, abnormal heart contraction arrested, abnormal heart contraction decreased rate, abnormal
Kopp et al., 2010	Fig. 1		kcnh6atb218/tb218	blood circulation decreased rate, abnormal heart contraction decreased rate, abnormal
	Fig. 11		kcnh6atb218/tb218	swimming decreased rate, abnormal
	Fig. 2		kcnh6atb218/tb218	blood circulation decreased rate, abnormal blood nucleate erythrocyte increased amount, abnormal dorsal aorta decreased diameter, abnormal
	Fig. 3		kcnh6atb218/tb218	parachordal vessel present, abnormal vertebral artery present, abnormal
	Fig. 4		kcnh6atb218/tb218	cell cycle decreased rate, abnormal whole organism decreased length, abnormal
	text only		kcnh6atb218/tb218	hatching delayed, abnormal
Chi et al., 2008	Fig. 7		kcnh6as290/s290; s878Tg ,   kcnh6as213/s213; s878Tg	calcium-mediated signaling disrupted, abnormal regulation of cardiac muscle contraction disrupted, abnormal
	Fig. Table 1		kcnh6as213/s213 ,   kcnh6as290/s290	cardiac ventricle non-contractile, abnormal
Hassel et al., 2008	Fig. 1		kcnh6am230/m230	heart contraction arrhythmic, abnormal pericardium edematous, abnormal
	Fig. 3		kcnh6am230/m230 + MO3-kcnh6a ,   WT + MO3-kcnh6a	heart contraction disrupted, abnormal
	Fig. 6		kcnh6am230/m230 ,   kcnh6am230/+	cardiac ventricle action potential, abnormal
	text only		kcnh6am230/m230	blood circulation disrupted, abnormal heart contraction disrupted, abnormal whole organism semi-viable, abnormal
Arnaout et al., 2007	Fig. 1		kcnh6as213/s213 ,   kcnh6as290/s290	blood circulation arrested, abnormal cardiac ventricle non-functional, abnormal pericardium edematous, abnormal
	Fig. 3		kcnh6as290/s290	cardiac ventricle action potential, abnormal
	Fig. 5		kcnh6as290/+	cardiac conduction increased duration, abnormal cardiac ventricle conductivity, abnormal
	text only		kcnh6as213/s213 ,   kcnh6as290/s290 ,   WT + MO1-kcnh6a ,   kcnh6as213/s213; s878Tg	cardiac ventricle non-contractile, abnormal cardiac ventricle non-functional, abnormal whole organism lethal (sensu genetics), abnormal
Phenotype Annotation (1994-2006)	Fig. for (m230)		kcnh6am230 (AB)	heart quality, abnormal
	Fig. for (s213)		kcnh6as213	heart quality, abnormal
	Fig. for (tb218)		kcnh6atb218/tb218	anatomical system quality, abnormal heart quality, abnormal
Chen et al., 1996	Table 1		kcnh6atb218/tb218	cardiac ventricle contractility, abnormal heart contraction irregular rhythm, abnormal
Stainier et al., 1996	Table 2		kcnh6am230/m230 (AB)	cardiac conduction rhythm quality, abnormal heart contractility, abnormal heart contraction disrupted, abnormal