Phenotype
|
Fish
|
Conditions
|
Figures
|
retinal ganglion cell decreased amount, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 8
from Yao et al., 2010
|
pharyngeal arch 3-7 decreased size, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 4
from Yao et al., 2010
|
somite cuboid, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 12
from Yao et al., 2010
|
somite border deformed, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 4 ,
Fig. 11
from Yao et al., 2010
|
retina disorganized, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 10
from Yao et al., 2010
|
skeletal muscle cell disorganized, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 12
from Yao et al., 2010
|
vertical myoseptum decreased length, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 12
from Yao et al., 2010
|
retinal neural layer morphology, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 8
from Yao et al., 2010
|
retina development in camera-type eye disrupted, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 10
from Yao et al., 2010
|
extension decreased length, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 4
from Yao et al., 2010
|
somite U-shaped, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 4 ,
Fig. 11 ,
Fig. 12
from Yao et al., 2010
|
whole organism anterior-posterior axis curved ventral, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 4
from Yao et al., 2010
|
somitogenesis disrupted, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 11
from Yao et al., 2010
|
post-vent region decreased flexibility, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 4
from Yao et al., 2010
|
skeletal muscle cell differentiation disrupted, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 12
from Yao et al., 2010
|
retina morphology, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 9
from Yao et al., 2010
|
vertical myoseptum structure, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 12
from Yao et al., 2010
|
retina decreased size, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 8
from Yao et al., 2010
|
retina apoptotic, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 9
from Yao et al., 2010
|
somite fused with somite, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 4
from Yao et al., 2010
|
somite posterior-most region decreased amount, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 11
from Yao et al., 2010
|
brain shape, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 4
from Yao et al., 2010
|
blood circulation disrupted, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 4
from Yao et al., 2010
|
retina layer formation disrupted, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 8
from Yao et al., 2010
|
eye decreased size, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 4
from Yao et al., 2010
|
head hypotrophic, abnormal
|
AB + MO1-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 4
from Yao et al., 2010
|
otolith decreased size, abnormal
|
AB + MO1-grhl2b + MO5-tp53
|
standard conditions
|
Fig. 3
from Han et al., 2011
|
post-vent region curved ventral, abnormal
|
AB + MO1-rer1 + MO5-tp53
|
standard conditions
|
Fig. 1
from Jurisch-Yaksi et al., 2013
|
whole organism anterior-posterior axis bent, abnormal
|
AB + MO1-rer1 + MO5-tp53
|
standard conditions
|
Fig. 1
from Jurisch-Yaksi et al., 2013
|
somite border deformed, abnormal
|
AB + MO2-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 5
from Yao et al., 2010
|
blood circulation disrupted, abnormal
|
AB + MO2-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 5
from Yao et al., 2010
|
post-vent region decreased flexibility, abnormal
|
AB + MO2-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 5
from Yao et al., 2010
|
extension decreased length, abnormal
|
AB + MO2-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 5
from Yao et al., 2010
|
pharyngeal arch 3-7 decreased size, abnormal
|
AB + MO2-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 5
from Yao et al., 2010
|
whole organism anterior-posterior axis curved ventral, abnormal
|
AB + MO2-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 5
from Yao et al., 2010
|
eye decreased size, abnormal
|
AB + MO2-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 5
from Yao et al., 2010
|
head hypotrophic, abnormal
|
AB + MO2-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 5
from Yao et al., 2010
|
brain shape, abnormal
|
AB + MO2-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 5
from Yao et al., 2010
|
somite fused with somite, abnormal
|
AB + MO2-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 5
from Yao et al., 2010
|
somite U-shaped, abnormal
|
AB + MO2-atoh8 + MO5-tp53
|
standard conditions
|
Fig. 5
from Yao et al., 2010
|
whole organism anterior-posterior axis bent, abnormal
|
AB + MO3-rer1 + MO5-tp53
|
standard conditions
|
Fig. 1
from Jurisch-Yaksi et al., 2013
|
post-vent region curved ventral, abnormal
|
AB + MO3-rer1 + MO5-tp53
|
standard conditions
|
Fig. 1
from Jurisch-Yaksi et al., 2013
|
whole organism col15a1b expression decreased amount, abnormal
|
AB/TU + MO3-col15a1b + MO5-tp53
|
standard conditions
|
Fig. 6
from Guillon et al., 2016
|
dorsal/ventral pattern formation disrupted, abnormal
|
TU + MO1-fbxl15 + MO5-tp53
|
standard conditions
|
Fig. 7
from Cui et al., 2011
|
trunk decreased length, abnormal
|
TU + MO1-fbxl15 + MO5-tp53
|
standard conditions
|
Fig. 7
from Cui et al., 2011
|
trunk increased curvature, abnormal
|
TU + MO1-fbxl15 + MO5-tp53
|
standard conditions
|
Fig. 7
from Cui et al., 2011
|
whole organism wholly dorsalized, abnormal
|
TU + MO1-fbxl15 + MO5-tp53
|
standard conditions
|
Fig. 7
from Cui et al., 2011
|
granulocyte decreased amount, abnormal
|
TU + MO5-etv5a + MO5-tp53
|
standard conditions
|
Fig. 2
from Chen et al., 2013
|
hematopoietic stem cell differentiation disrupted, abnormal
|
TU + MO5-etv5a + MO5-tp53
|
standard conditions
|
Fig. 2
from Chen et al., 2013
|
hematopoietic stem cell decreased amount, abnormal
|
TU + MO5-etv5a + MO5-tp53
|
standard conditions
|
Fig. 2
from Chen et al., 2013
|
erythroid lineage cell decreased amount, abnormal
|
TU + MO5-etv5a + MO5-tp53
|
standard conditions
|
Fig. 2
from Chen et al., 2013
|
hemopoiesis disrupted, abnormal
|
TU + MO5-etv5a + MO5-tp53
|
standard conditions
|
Fig. 2
from Chen et al., 2013
|
hemangioblast cell differentiation disrupted, abnormal
|
TU + MO5-etv5a + MO5-tp53
|
standard conditions
|
Fig. 2
from Chen et al., 2013
|
common lymphoid progenitor decreased amount, abnormal
|
TU + MO5-etv5a + MO5-tp53
|
standard conditions
|
Fig. 2
from Chen et al., 2013
|
skeletal muscle refractivity, abnormal
|
WIK + MO1-hspb9l + MO5-tp53
|
standard conditions
|
Figure 6
from Klüver et al., 2011
|
slow muscle cell myosin filament disorganized, abnormal
|
WIK + MO1-hspb9l + MO5-tp53
|
standard conditions
|
Figure 6
from Klüver et al., 2011
|
skeletal muscle refractivity, abnormal
|
WIK + MO2-hspb9l + MO5-tp53
|
standard conditions
|
Figure 6
from Klüver et al., 2011
|
slow muscle cell myosin filament disorganized, abnormal
|
WIK + MO2-hspb9l + MO5-tp53
|
standard conditions
|
Figure 6
from Klüver et al., 2011
|
whole organism sod1 expression decreased amount, abnormal
|
WT + MO1-atp7a + MO5-tp53
|
standard conditions
|
Fig. 1
from Chen et al., 2011
|
head apoptotic, abnormal
|
WT + MO1-cenph + MO2-cenph + MO5-tp53
|
standard conditions
|
Fig. 3
from Zhao et al., 2010
|
spinal cord opaque, abnormal
|
WT + MO1-cenph + MO2-cenph + MO5-tp53
|
standard conditions
|
Fig. 3
from Zhao et al., 2010
|
integument rough, abnormal
|
WT + MO1-cenph + MO2-cenph + MO5-tp53
|
standard conditions
|
Fig. 3
from Zhao et al., 2010
|
trunk curved dorsal, abnormal
|
WT + MO1-cenph + MO2-cenph + MO5-tp53
|
standard conditions
|
Fig. 3
from Zhao et al., 2010
|
spinal cord apoptotic, abnormal
|
WT + MO1-cenph + MO2-cenph + MO5-tp53
|
standard conditions
|
Fig. 3
from Zhao et al., 2010
|
head opaque, abnormal
|
WT + MO1-cenph + MO2-cenph + MO5-tp53
|
standard conditions
|
Fig. 3
from Zhao et al., 2010
|
eye decreased size, abnormal
|
WT + MO1-cenph + MO2-cenph + MO5-tp53
|
standard conditions
|
Fig. 3
from Zhao et al., 2010
|
roof of mouth development disrupted, abnormal
|
WT + MO1-faf1 + MO5-tp53
|
standard conditions
|
Fig. 2
from Ghassibe-Sabbagh et al., 2011
|
mouth open, abnormal
|
WT + MO1-faf1 + MO5-tp53
|
standard conditions
|
Fig. 2
from Ghassibe-Sabbagh et al., 2011
|
neural tube morphology, abnormal
|
WT + MO1-fbxl18 + MO5-tp53
|
standard conditions
|
Fig. 5
from Lee et al., 2017
|
sclerotome structure, abnormal
|
WT + MO1-fbxl18 + MO5-tp53
|
standard conditions
|
Fig. 5
from Lee et al., 2017
|
trunk truncated, abnormal
|
WT + MO1-iqgap3 + MO5-tp53
|
standard conditions
|
Fig. 5
from Fang et al., 2015
|
somite shape, abnormal
|
WT + MO1-iqgap3 + MO5-tp53
|
standard conditions
|
Fig. 5
from Fang et al., 2015
|
somite increased size, abnormal
|
WT + MO1-iqgap3 + MO5-tp53
|
standard conditions
|
Fig. 5
from Fang et al., 2015
|
whole organism anterior-posterior axis truncated, abnormal
|
WT + MO1-iqgap3 + MO5-tp53
|
standard conditions
|
Fig. 5
from Fang et al., 2015
|
convergent extension involved in gastrulation disrupted, abnormal
|
WT + MO1-iqgap3 + MO5-tp53
|
standard conditions
|
Fig. 5
from Fang et al., 2015
|
whole organism anterior-posterior axis increased width, abnormal
|
WT + MO1-iqgap3 + MO5-tp53
|
standard conditions
|
Fig. 5
from Fang et al., 2015
|
caudal fin curved, abnormal
|
WT + MO1-ppp4cb + MO5-tp53
|
standard conditions
|
Fig. 1
from Jia et al., 2012
|
trunk posterior region decreased size, abnormal
|
WT + MO1-ppp4cb + MO5-tp53
|
standard conditions
|
Fig. 1
from Jia et al., 2012
|
caudal fin decreased thickness, abnormal
|
WT + MO1-ppp4cb + MO5-tp53
|
standard conditions
|
Fig. 1
from Jia et al., 2012
|
whole organism wholly dorsalized, abnormal
|
WT + MO1-ppp4cb + MO5-tp53
|
standard conditions
|
Fig. 1
from Jia et al., 2012
|
tail bud malformed, exacerbated
|
WT + MO1-txnb + MO5-tp53
|
chemical treatment by environment: methylmercury chloride
|
Fig. 5
from Yang et al., 2019
|
ventricular system hydrocephalic, exacerbated
|
WT + MO1-txnb + MO5-tp53
|
chemical treatment by environment: methylmercury chloride
|
Fig. 5
from Yang et al., 2019
|
midbrain decreased size, abnormal
|
WT + MO1-txnb + MO5-tp53
|
control
|
Fig. 4
from Yang et al., 2019
|
third ventricle hydrocephalic, abnormal
|
WT + MO1-txnb + MO5-tp53
|
control
|
Fig. 4
from Yang et al., 2019
|
ventricular zone apoptotic process increased occurrence, abnormal
|
WT + MO1-txnb + MO5-tp53
|
control
|
Fig. 4
from Yang et al., 2019
|
fin malformed, exacerbated
|
WT + MO1-txnb + MO5-tp53
|
chemical treatment by environment: methylmercury chloride
|
Fig. 5
from Yang et al., 2019
|
ventricular system hydrocephalic, abnormal
|
WT + MO1-txnb + MO5-tp53
|
control
|
Fig. 4
from Yang et al., 2019
|
midbrain hindbrain boundary malformed, abnormal
|
WT + MO1-txnb + MO5-tp53
|
control
|
Fig. 4
from Yang et al., 2019
|
medulla oblongata decreased size, abnormal
|
WT + MO1-txnb + MO5-tp53
|
control
|
Fig. 4
from Yang et al., 2019
|
pericardium edematous, exacerbated
|
WT + MO1-txnb + MO5-tp53
|
chemical treatment by environment: methylmercury chloride
|
Fig. 5
from Yang et al., 2019
|
mouth open, abnormal
|
WT + MO2-faf1 + MO5-tp53
|
standard conditions
|
Fig. 2
from Ghassibe-Sabbagh et al., 2011
|
roof of mouth development disrupted, abnormal
|
WT + MO2-faf1 + MO5-tp53
|
standard conditions
|
Fig. 2
from Ghassibe-Sabbagh et al., 2011
|
whole organism decreased length, abnormal
|
WT + MO2-loxa + MO5-tp53
|
standard conditions
|
Fig. 4
from Reynaud et al., 2008
|
whole organism morphology, abnormal
|
WT + MO2-loxa + MO5-tp53
|
standard conditions
|
Fig. 4
from Reynaud et al., 2008
|
optic tectum structure, abnormal
|
WT + MO2-mmp2 + MO5-tp53
|
standard conditions
|
Fig. S2
from Janssens et al., 2013
|
retinal ganglion cell axon guidance disrupted, abnormal
|
WT + MO2-mmp2 + MO5-tp53
|
standard conditions
|
Fig. S2
from Janssens et al., 2013
|
eye decreased diameter, abnormal
|
WT + MO2-mmp2 + MO5-tp53
|
standard conditions
|
Fig. S2
from Janssens et al., 2013
|
whole organism decreased length, abnormal
|
WT + MO2-mmp2 + MO5-tp53
|
standard conditions
|
Fig. S2
from Janssens et al., 2013
|
whole organism wholly dorsalized, abnormal
|
WT + MO2-ppp4cb + MO5-tp53
|
standard conditions
|
Fig. 1
from Jia et al., 2012
|
caudal fin curved, abnormal
|
WT + MO2-ppp4cb + MO5-tp53
|
standard conditions
|
Fig. 1
from Jia et al., 2012
|
trunk posterior region decreased size, abnormal
|
WT + MO2-ppp4cb + MO5-tp53
|
standard conditions
|
Fig. 1
from Jia et al., 2012
|
caudal fin decreased thickness, abnormal
|
WT + MO2-ppp4cb + MO5-tp53
|
standard conditions
|
Fig. 1
from Jia et al., 2012
|
yolk swollen, abnormal
|
WT + MO2-prmt8b + MO5-tp53
|
standard conditions
|
Fig. S1
from Lin et al., 2013
|
brain malformed, abnormal
|
WT + MO2-prmt8b + MO5-tp53
|
standard conditions
|
Fig. S1
from Lin et al., 2013
|
eye decreased size, abnormal
|
WT + MO2-prmt8b + MO5-tp53
|
standard conditions
|
Fig. S1
from Lin et al., 2013
|
heart edematous, abnormal
|
WT + MO2-prmt8b + MO5-tp53
|
standard conditions
|
Fig. S1
from Lin et al., 2013
|
post-vent region curved, abnormal
|
WT + MO2-prmt8b + MO5-tp53
|
standard conditions
|
Fig. S1
from Lin et al., 2013
|
neural tube morphology, abnormal
|
WT + MO2-tgfb2 + MO5-tp53
|
standard conditions
|
Fig. 5
from Lee et al., 2017
|
sclerotome structure, abnormal
|
WT + MO2-tgfb2 + MO5-tp53
|
standard conditions
|
Fig. 5
from Lee et al., 2017
|
neural tube morphology, abnormal
|
WT + MO2-tle3a + MO5-tp53
|
standard conditions
|
Fig. 5
from Lee et al., 2017
|
sclerotome structure, abnormal
|
WT + MO2-tle3a + MO5-tp53
|
standard conditions
|
Fig. 5
from Lee et al., 2017
|
trunk posterior region decreased size, abnormal
|
WT + MO3-ppp4ca + MO5-tp53
|
standard conditions
|
Fig. 1
from Jia et al., 2012
|
caudal fin decreased thickness, abnormal
|
WT + MO3-ppp4ca + MO5-tp53
|
standard conditions
|
Fig. 1
from Jia et al., 2012
|
caudal fin curved, abnormal
|
WT + MO3-ppp4ca + MO5-tp53
|
standard conditions
|
Fig. 1
from Jia et al., 2012
|
whole organism wholly dorsalized, abnormal
|
WT + MO3-ppp4ca + MO5-tp53
|
standard conditions
|
Fig. 1
from Jia et al., 2012
|
retinal ganglion cell decreased amount, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 6
from Janssens et al., 2013
|
retinal ganglion cell axon guidance disrupted, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 8
from Janssens et al., 2013
|
retina has fewer parts of type amacrine cell, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 6
from Janssens et al., 2013
|
retina layer formation disrupted, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 6
from Janssens et al., 2013
|
cranial nerve II hypoplastic, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 6
from Janssens et al., 2013
|
cell proliferation involved in compound eye morphogenesis increased occurrence, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 7
from Janssens et al., 2013
|
neural retina development delayed, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 6
from Janssens et al., 2013
|
retina lacks all parts of type amacrine cell, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 6
from Janssens et al., 2013
|
Muller cell decreased amount, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 6
from Janssens et al., 2013
|
Meckel's cartilage malformed, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 4
from Janssens et al., 2013
|
retina layer formation delayed, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 6
from Janssens et al., 2013
|
eye decreased size, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 5
from Janssens et al., 2013
|
retina morphology, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 6
from Janssens et al., 2013
|
ciliary marginal zone increased size, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 7
from Janssens et al., 2013
|
ceratohyal cartilage malformed, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 4
from Janssens et al., 2013
|
retina proliferative region increased size, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 7
from Janssens et al., 2013
|
retinal cone cell decreased amount, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 6
from Janssens et al., 2013
|
eye decreased diameter, abnormal
|
WT + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 5 ,
Fig. 9
from Janssens et al., 2013
|
caudal fin decreased thickness, abnormal
|
WT + MO4-ppp4ca + MO5-tp53
|
standard conditions
|
Fig. 1
from Jia et al., 2012
|
trunk posterior region decreased size, abnormal
|
WT + MO4-ppp4ca + MO5-tp53
|
standard conditions
|
Fig. 1
from Jia et al., 2012
|
caudal fin curved, abnormal
|
WT + MO4-ppp4ca + MO5-tp53
|
standard conditions
|
Fig. 1
from Jia et al., 2012
|
whole organism wholly dorsalized, abnormal
|
WT + MO4-ppp4ca + MO5-tp53
|
standard conditions
|
Fig. 1
from Jia et al., 2012
|
eye decreased size, abnormal
|
WT + MO5-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 5
from Janssens et al., 2013
|
eye decreased diameter, abnormal
|
WT + MO5-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 5
from Janssens et al., 2013
|
cranial nerve II decreased thickness, abnormal
|
rw021Tg + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 8
from Janssens et al., 2013
|
neural retina development delayed, abnormal
|
rw021Tg + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 7
from Janssens et al., 2013
|
neural retina development delayed, abnormal
|
rw021Tg + MO5-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 7
from Janssens et al., 2013
|
neural crest cell migration disrupted, abnormal
|
y1Tg + MO1-faf1 + MO5-tp53
|
standard conditions
|
Fig. 2
from Ghassibe-Sabbagh et al., 2011
|
pharyngeal arch cranial neural crest mislocalised, abnormal
|
y1Tg + MO1-faf1 + MO5-tp53
|
standard conditions
|
Fig. 2
from Ghassibe-Sabbagh et al., 2011
|
neural crest cell migration disrupted, abnormal
|
y1Tg + MO2-faf1 + MO5-tp53
|
standard conditions
|
Fig. 2
from Ghassibe-Sabbagh et al., 2011
|
pharyngeal arch cranial neural crest mislocalised, abnormal
|
y1Tg + MO2-faf1 + MO5-tp53
|
standard conditions
|
Fig. 2
from Ghassibe-Sabbagh et al., 2011
|
posterior lateral line neuromast decreased amount, abnormal
|
zf106Tg + MO1-smad5 + MO5-tp53
|
standard conditions
|
Fig. 5
from Xing et al., 2015
|
posterior lateral line neuromast far from posterior lateral line neuromast, abnormal
|
zf106Tg + MO1-smad5 + MO5-tp53
|
standard conditions
|
Fig. 5
from Xing et al., 2015
|
posterior lateral line neuromast far from posterior lateral line neuromast, abnormal
|
zf106Tg + MO4-tgfb1a + MO5-tp53
|
standard conditions
|
Fig. 1
from Xing et al., 2015
|
posterior lateral line neuromast decreased amount, abnormal
|
zf106Tg + MO4-tgfb1a + MO5-tp53
|
standard conditions
|
Fig. 1
from Xing et al., 2015
|
posterior lateral line neuromast irregular spatial pattern, abnormal
|
zf106Tg + MO4-tgfb1a + MO5-tp53
|
standard conditions
|
Fig. 1
from Xing et al., 2015
|
head apoptotic, abnormal
|
cenphtsu055Gt/tsu055Gt + MO5-tp53
|
standard conditions
|
Fig. 5
from Zhao et al., 2010
|
trunk curved dorsal, abnormal
|
cenphtsu055Gt/tsu055Gt + MO5-tp53
|
standard conditions
|
Fig. 5
from Zhao et al., 2010
|
brain apoptotic, abnormal
|
cenphtsu055Gt/tsu055Gt + MO5-tp53
|
standard conditions
|
Fig. 5
from Zhao et al., 2010
|
spinal cord opaque, abnormal
|
cenphtsu055Gt/tsu055Gt + MO5-tp53
|
standard conditions
|
Fig. 5
from Zhao et al., 2010
|
eye decreased size, abnormal
|
cenphtsu055Gt/tsu055Gt + MO5-tp53
|
standard conditions
|
Fig. 5
from Zhao et al., 2010
|
head opaque, abnormal
|
cenphtsu055Gt/tsu055Gt + MO5-tp53
|
standard conditions
|
Fig. 5
from Zhao et al., 2010
|
spinal cord apoptotic, abnormal
|
cenphtsu055Gt/tsu055Gt + MO5-tp53
|
standard conditions
|
Fig. 5
from Zhao et al., 2010
|
vasculature hemorrhagic, abnormal
|
gbf1tsu3994/tsu3994 + MO5-tp53
|
standard conditions
|
Fig. 9
from Chen et al., 2017
|
eye decreased diameter, abnormal
|
WT + MO1-mmp14b + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 9
from Janssens et al., 2013
|
retinal ganglion cell axon guidance disrupted, abnormal
|
WT + MO2-mmp2 + MO4-mmp14a + MO5-tp53
|
standard conditions
|
Fig. 10
from Janssens et al., 2013
|