Fig. S4
- ID
- ZDB-FIG-170425-19
- Publication
- Delfino-Machín et al., 2017 - Sox10 contributes to the balance of fate choice in dorsal root ganglion progenitors
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Oligodendrocyte differentiation, but not specification, is disrupted in baz1 mutants. A,B) Oligodendrocyte precursors at 48 hpf in trunk spinal cord express sox10 and are indistinguishable in number and distribution in WT (A) and baz1 mutant (B). C,D) sox10 expression in dispersing oligodendrocyte of hindbrain; note that numbers and dorsoventral distribution strongly resemble wild-type siblings. E,F) sox10 expression in oligodendrocyte progenitors in the ventralmost spinal cord are unaffected at 72 hpf; region of somites 7–11 is shown. G,H) Oligodendrocyte differentiation is abnormal as shown by strongly decreased mbp expression in hindbrain of 72 hpf embryo. |