FIGURE

Fig. S3

ID

ZDB-FIG-151009-31

Publication

Jia et al., 2015 - Mutation of kri1l causes definitive hematopoiesis failure via PERK-dependent excessive autophagy induction

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Fig. S3

WISH analysis of primitive hematopoiesis and vascular morphogenesis. WT or mutant^cas002 embryos were fixed at 22 hpf, followed by WISH analysis of scl, gata1, spi1, mpo expression. Expression of scl (A-B), gata1 (C-D), spi1 (E-F) and mpo (G-H) show no significant difference between wild-type siblings and mutants at 22 hpf. Insets in E-F show dorsal views of the head staining region. Embryos were fixed at 26 dpf, followed by WISH analysis of expression pattern of kdr. Expression of kdr (I-J), which marks vascular endothelial, had no significant difference between siblings and mutants at 26 hpf. The number and percentage of het-het incross embryos with the in situ pattern are listed at the bottom of the Mutant^cas002 panels.

Expression Data

Expression Detail

Antibody Labeling

Phenotype Data

Fish:	kri1^{cas002/cas002}
Observed In:	primitive hemopoiesis vasculogenesis
Stage:	26+ somites

Phenotype Detail

Acknowledgments

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