FIGURE

Fig. 3

ID

ZDB-FIG-151002-8

Publication

Liu et al., 2015 - Apoc2 loss-of-function zebrafish mutant as a genetic model of hyperlipidemia

Other Figures

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Fig. 3

Development and angiogenesis in apoc2 mutant larvae. (A-C) Morphology of WT (n=11) and apoc2 mutant (mut, n=11) embryos at 30hpf. Results are mean±s.e.m.; ***P<0.001 (Student′s t-test). (D-G) Morphology of WT (n=11) and apoc2 mutant (n=10) embryos at 3.5dpf. Results are mean±s.e.m.; *P<0.05; **P<0.01; ***P<0.001 (Student′s t-test). The blue, red and yellow lengths highlighted in D were used to delineate the size of yolk, yolk extension and the body length, respectively. (H) Angiogenesis in fli1:EGFP WT and apoc2 mutant embryos at 28hpf (a,a′), 52hpf (b,b′) and 80hpf (c,c′). The numbers in the figure indicate the number of animals exhibiting the illustrated phenotype and the total number of animals. Scale bars: 200µm (A,D); 100µm (H).

Expression Data

Gene:	EGFP
Fish:	y1Tg apoc2^sd38/sd38; y1Tg
Anatomical Terms:	endothelial cell intersegmental vessel subintestinal vein trunk vasculature vasculature
Stage Range:	Prim-5 to Protruding-mouth

Expression Detail

Antibody Labeling

Phenotype Data

Fish:	apoc2^sd38/sd38 apoc2^sd38/sd38; y1Tg
Observed In:	ball extension intersegmental vessel angiogenesis subintestinal vein angiogenesis whole organism
Stage Range:	Prim-15 to Protruding-mouth

Phenotype Detail

Acknowledgments

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