FIGURE
Fig. 5
- ID
- ZDB-FIG-100114-29
- Publication
- Codina et al., 2010 - Loss of Smyhc1 or Hsp90alpha1 function results in different effects on myofibril organization in skeletal muscles of zebrafish embryos
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Fig. 5
Knockdown of smyhc1 expression or hsp90α1 mutation resulted in defective thin filament organization in skeletal muscles of zebrafish embryos. A–C. Anti-α-actin antibody staining shows the organization of thin filaments in slow muscles of control (A), smyhc1 knockdown (B), or slotu44c mutant (C) embryos at 48 hpf. D, F. Anti-α-actin antibody staining shows the organization of thin filaments in fast muscles of control (D), smyhc1 knockdown (E), or slotu44c mutant (F) embryos at 72 hpf. Scale bar = 25 μm in A. |
Expression Data
| Antibody: | |
|---|---|
| Fish: | |
| Knockdown Reagent: | |
| Anatomical Terms: | |
| Stage Range: | Long-pec to Protruding-mouth |
Expression Detail
Antibody Labeling
Phenotype Data
| Fish: | |
|---|---|
| Knockdown Reagent: | |
| Observed In: | |
| Stage Range: | Long-pec to Protruding-mouth |
Phenotype Detail
Acknowledgments
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Full text @ PLoS One