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Fig. 3 Migration of the PLLP is defective following inactivation of the kal1a, but not kal1b, gene as visualized by whole-mount in situ detection of cxcr4b RNAs at 28 hpf. Wild-type (WT) embryo (A), MO kal1a (0.5 mM) (B) and mmMO kal1a morphants (0.5 mM) (C), embryo injected with MO kal1a-insensitive kal1a RNA (1 μM) alone (D) and MO kal1a (0.5 mM) morphant injected with MO kal1a-insensitive kal1a RNA (1 μM) (E). Arrows indicate the leading edge of the migrating PLLP.
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Reprinted from Developmental Biology, 320(2), Yanicostas, C., Ernest, S., Dayraud, C., Petit, C., and Soussi-Yanicostas, N., Essential requirement for zebrafish anosmin-1a in the migration of the posterior lateral line primordium, 469-479, Copyright (2008) with permission from Elsevier. Full text @ Dev. Biol.