FIGURE

Fig. 3

ID

ZDB-FIG-140313-16

Publication

Hild et al., 1999 - The smad5 mutation somitabun blocks Bmp2b signaling during early dorsoventral patterning of the zebrafish embryo

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Fig. 3

Later phenotypes of sbntc24 mutant embryos, 36 hpf, lateral view. (A) Wild type; (B) dominant maternal effect of sbn, causing strong C4 dorsalization. (C) Dominant zygotic effect of sbn, causing weak C1 dorsalization (arrow to ventral tail fin deficiency). (D) sbn,swr double heterozygote from a cross of a swr heterozygous female and a sbn heterozygous male, characterized by a loss of the ventral tail fin and a wound-up tail (C3, arrow), indicating a significantly stronger dorsalization than in C. (E-H) The maternal effect of sbn in the background of different sbn genotypes, 48 hpf, lateral view; (E-G) Embryos from a cross of two sbn heterozygous parents; (H) embryo from a cross of a sbn homozygous female with a heterozygous male. Both crosses led to offspring with 100% C4 dorsalization. The maternal and zygotic genotypes of individual embryos are indicated. The zygotic genotype was determined by tetra-PCR after photography.

Expression Data

Expression Detail

Antibody Labeling

Phenotype Data

Fish:	smad5^dtc24/dtc24 smad5^dtc24/dtc24 smad5^dtc24/+ smad5^dtc24/+ smad5^dtc24 smad5^dtc24 bmp2b^ta72a/+; smad5^dtc24/+
Observed In:	caudal fin whole organism
Stage Range:	Prim-5 to Long-pec

Phenotype Detail

Acknowledgments

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