Publication	Data		Fish	Anatomy
Miyake et al., 2017	Fig. 3		WT + MO2-vwc2 ,   WT + MO1-vwc2	brain morphogenesis decreased process quality, abnormal tectal ventricle malformed, abnormal telencephalon diencephalon boundary malformed, abnormal whole organism vwc2 expression absent, abnormal whole organism vwc2 expression decreased amount, abnormal
	Fig. 4		WT + MO1-vwc2	brain BMP signaling pathway increased occurrence, abnormal brain dorsal region ab1-smad labeling increased amount, abnormal
	Fig. 6		WT + MO1-vwc2	dorsal telencephalon forebrain morphogenesis decreased process quality, abnormal ventral telencephalon dlx2a expression decreased amount, abnormal ventral telencephalon emx1 expression mislocalised, abnormal ventral telencephalon tbr1b expression mislocalised, abnormal ventral telencephalon forebrain morphogenesis decreased process quality, abnormal ventral thalamus dlx2a expression decreased amount, abnormal ventral thalamus forebrain morphogenesis decreased process quality, abnormal
	Fig. 7		WT + MO1-vwc2	brain astrocyte glula expression increased amount, abnormal brain astrocyte development increased occurrence, abnormal brain oligodendrocyte plp1a expression decreased amount, abnormal brain oligodendrocyte development decreased occurrence, abnormal diencephalon GABAergic neuron ascl1a expression decreased amount, abnormal diencephalon neuron differentiation decreased process quality, abnormal diencephalon ventral region neurog1 expression decreased amount, abnormal forebrain GABAergic neuron differentiation decreased process quality, abnormal nucleus of the tract of the postoptic commissure GABAergic neuron gad1b expression decreased amount, abnormal ventral telencephalon GABAergic neuron ascl1a expression decreased amount, abnormal ventral telencephalon GABAergic neuron gad1b expression decreased amount, abnormal ventral telencephalon neuron differentiation decreased process quality, abnormal
	Fig. 8		WT + MO1-vwc2	anterior commissure central nervous system projection neuron axonogenesis decreased occurrence, abnormal forebrain lacks all parts of type anterior commissure, abnormal forebrain lacks all parts of type postoptic commissure, abnormal forebrain lacks all parts of type supraoptic tract, abnormal postoptic commissure central nervous system projection neuron axonogenesis decreased occurrence, abnormal supraoptic tract central nervous system projection neuron axonogenesis decreased occurrence, abnormal
	Fig. 9		WT + MO1-vwc2	diencephalon sema3d expression decreased amount, abnormal telencephalon ntn1a expression increased amount, abnormal ventral thalamus ntn1a expression increased amount, abnormal