PUBLICATION

Long noncoding RNA HIKER regulates erythropoiesis in Monge's disease via CSNK2B

Authors
Azad, P., Zhou, D., Tu, H.C., Villafuerte, F.C., Traver, D., Rana, T.M., Haddad, G.G.
ID
ZDB-PUB-230407-46
Date
2023
Source
The Journal of Clinical Investigation   133(11): (Journal)
Registered Authors
Traver, David
Keywords
Genetics, Hematology, Hypoxia, Noncoding RNAs
MeSH Terms
  • Altitude Sickness*/genetics
  • Animals
  • Casein Kinase II*/metabolism
  • Chronic Disease
  • Erythropoiesis/genetics
  • Humans
  • Hypoxia/genetics
  • Polycythemia*
  • RNA, Long Noncoding*/genetics
  • Zebrafish/genetics
PubMed
37022795 Full text @ Journal of Clin. Invest.
Abstract
Excessive Erythrocytosis (EE) is a major hallmark of patients suffering from chronic mountain sickness (CMS, Monge's disease) and is responsible for major morbidity and even mortality in early adulthood. We took advantage of unique populations, one living at high altitude (Peru) showing EE, while another population, at the same altitude and region, shows no evidence of EE (non-CMS). Through RNA-seq, we identified and validated the function of a group of long non-coding RNA (lncRNAs) that regulate erythropoiesis in Monge's disease but not in the non-CMS population. Among these lncRNAs is HIKER (Hypoxia Induced Kinase-mediated Erythropoietic Regulator)/LINC02228 which we showed plays a critical role in erythropoiesis in CMS cells. Under hypoxia, HIKER modulated CSNK2B (the regulatory subunit of Casein kinase 2). A down-regulation of HIKER down-regulated CSNK2B, remarkably reducing erythropoiesis (<70% reduction of BFUs); furthermore, an up-regulation of CSNK2B on the background of HIKER down-regulation rescued erythropoiesis defects. Pharmacologic inhibition of CSNK2B drastically reduced erythroid colonies (50-75% reduction in BFU colonies) and knock-down of CSNK2B in zebrafish lead to a defect in hemoglobinization (<97% morphants show reduction in hemoglobin levels). We conclude that HIKER regulates erythropoiesis in Monge's disease and acts through at least one specific target, CSNK2B, a casein kinase.
Genes / Markers
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Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
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Mapping