PUBLICATION

Timely Schwann cell division drives peripheral myelination in vivo via Laminin/cAMP pathway

Authors
Mikdache, A., Boueid, M.J., Lesport, E., Delespierre, B., Loisel-Duwattez, J., Degerny, C., Tawk, M.
ID
ZDB-PUB-220809-10
Date
2022
Source
Development (Cambridge, England)   149(17): (Journal)
Registered Authors
Boueid, Marie-Jose, Degerny, Cindy, Delespierre, Brigitte, Lesport, Emilie, Tawk, Marcel
Keywords
Laminin/cAMP, MCPH, Mitotic spindle, Myelin, Schwann cells, Zebrafish
MeSH Terms
  • Axons/metabolism
  • Cell Division/genetics
  • Cells, Cultured
  • Laminin*/genetics
  • Laminin*/metabolism
  • Myelin Sheath/metabolism
  • Schwann Cells*/metabolism
PubMed
35938454 Full text @ Development
Abstract
Schwann cells (SC) migrate along peripheral axons and divide intensively to generate the right number of cells prior to axonal ensheathment; however, little is known regarding the temporal and molecular control of their division and its impact on myelination. We report that Sil, a spindle pole protein associated with autosomal recessive primary microcephaly (MCPH), is required for temporal mitotic exit of SC. In sil-deficient cassiopeia (csp-/-) mutants, SC fail to radially sort and myelinate peripheral axons. Elevation of cAMP, but not Rac1 activity in csp-/- restores myelin ensheathment. Most importantly, we show a significant decrease in Laminin expression within csp-/- posterior lateral line nerve and that forcing Laminin2 expression in csp-/- fully restores SC ability to myelinate. Thus, we unravel a novel and essential role for timely SC division in mediating Laminin expression to orchestrate radial sorting and peripheral myelination in vivo.
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