PUBLICATION

Cilia-localized LKB1 regulates chemokine signaling, macrophage recruitment, and tissue homeostasis in the kidney

Authors
Viau, A., Bienaimé, F., Lukas, K., Todkar, A.P., Knoll, M., Yakulov, T.A., Hofherr, A., Kretz, O., Helmstädter, M., Reichardt, W., Braeg, S., Aschman, T., Merkle, A., Pfeifer, D., Dumit, V.I., Gubler, M.C., Nitschke, R., Huber, T.B., Terzi, F., Dengjel, J., Grahammer, F., Köttgen, M., Busch, H., Boerries, M., Walz, G., Triantafyllopoulou, A., Kuehn, E.W.
ID
ZDB-PUB-190418-20
Date
2018
Source
The EMBO journal   37(15): (Journal)
Registered Authors
Keywords
cilia, macrophages, nephronophthisis, polycystic kidney disease
MeSH Terms
  • Animals
  • Carrier Proteins/metabolism
  • Cell Line
  • Chemokine CCL2/metabolism*
  • Cilia/pathology*
  • Dogs
  • Epithelial Cells/metabolism
  • Female
  • HEK293 Cells
  • Humans
  • Kidney Diseases, Cystic/congenital*
  • Kidney Diseases, Cystic/pathology
  • Kidney Tubules/cytology
  • Kidney Tubules/pathology
  • Macrophages/metabolism
  • Madin Darby Canine Kidney Cells
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Phagocytosis/physiology
  • Polycystic Kidney, Autosomal Dominant/genetics
  • Polycystic Kidney, Autosomal Dominant/pathology*
  • Protein Kinase C/genetics*
  • Protein Kinase C/metabolism
  • Protein Serine-Threonine Kinases/genetics*
  • Protein Serine-Threonine Kinases/metabolism
  • Zebrafish
PubMed
29925518 Full text @ EMBO J.
Abstract
Polycystic kidney disease (PKD) and other renal ciliopathies are characterized by cysts, inflammation, and fibrosis. Cilia function as signaling centers, but a molecular link to inflammation in the kidney has not been established. Here, we show that cilia in renal epithelia activate chemokine signaling to recruit inflammatory cells. We identify a complex of the ciliary kinase LKB1 and several ciliopathy-related proteins including NPHP1 and PKD1. At homeostasis, this ciliary module suppresses expression of the chemokine CCL2 in tubular epithelial cells. Deletion of LKB1 or PKD1 in mouse renal tubules elevates CCL2 expression in a cell-autonomous manner and results in peritubular accumulation of CCR2+ mononuclear phagocytes, promoting a ciliopathy phenotype. Our findings establish an epithelial organelle, the cilium, as a gatekeeper of tissue immune cell numbers. This represents an unexpected disease mechanism for renal ciliopathies and establishes a new model for how epithelial cells regulate immune cells to affect tissue homeostasis.
Genes / Markers
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Human Disease / Model
Sequence Targeting Reagents
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