PUBLICATION

Probiotic treatment reduces appetite and glucose level in the zebrafish model

Authors
Falcinelli, S., Rodiles, A., Unniappan, S., Picchietti, S., Gioacchini, G., Merrifield, D.L., Carnevali, O.
ID
ZDB-PUB-160106-12
Date
2016
Source
Scientific Reports   6: 18061 (Journal)
Registered Authors
Carnevali, Oliana, Unniappan, Suraj
Keywords
Metabolic engineering, Microbiome, Transcriptional regulatory elements
MeSH Terms
  • Animals
  • Appetite*
  • Fatty Acids/metabolism
  • Gastrointestinal Absorption
  • Gastrointestinal Microbiome
  • Gastrointestinal Tract/metabolism
  • Gastrointestinal Tract/microbiology
  • Gastrointestinal Tract/ultrastructure
  • Gene Expression
  • Glucose/metabolism*
  • Larva/genetics
  • Larva/metabolism
  • Probiotics/administration & dosage*
  • Zebrafish/physiology*
PubMed
26727958 Full text @ Sci. Rep.
Abstract
The gut microbiota regulates metabolic pathways that modulate the physiological state of hunger or satiety. Nutrients in the gut stimulate the release of several appetite modulators acting at central and peripheral levels to mediate appetite and glucose metabolism. After an eight-day exposure of zebrafish larvae to probiotic Lactobacillus rhamnosus, high-throughput sequence analysis evidenced the ability of the probiotic to modulate the microbial composition of the gastrointestinal tract. These changes were associated with a down-regulation and up-regulation of larval orexigenic and anorexigenic genes, respectively, an up-regulation of genes related to glucose level reduction and concomitantly reduced appetite and body glucose level. BODIPY-FL-pentanoic-acid staining revealed higher short chain fatty acids levels in the intestine of treated larvae. These results underline the capability of the probiotic to modulate the gut microbiota community and provides insight into how the probiotic interacts to regulate a novel gene network involved in glucose metabolism and appetite control, suggesting a possible role for L. rhamnosus in the treatment of impaired glucose tolerance and food intake disorders by gut microbiota manipulation.
Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping