PUBLICATION

Efficient CRISPR/Cas9 genome editing with low off-target effects in zebrafish

Authors
Hruscha, A., Krawitz, P., Rechenberg, A., Heinrich, V., Hecht, J., Haass, C., and Schmid, B.
ID
ZDB-PUB-140102-5
Date
2013
Source
Development (Cambridge, England)   140(24): 4982-4987 (Journal)
Registered Authors
Haass, Christian, Hruscha, Alexander, Schmid, Bettina
Keywords
C9orf72, CRISPR/Cas9, genome editing, Knock in, Off-target, Zebrafish, Tardbp
MeSH Terms
  • Animals
  • Base Sequence
  • Caspase 9/genetics*
  • Clustered Regularly Interspaced Short Palindromic Repeats/genetics*
  • Gene Knock-In Techniques
  • Genetic Engineering/methods*
  • Genome/genetics*
  • Mutagenesis
  • Oligonucleotides, Antisense/genetics
  • Sequence Analysis, DNA
  • Zebrafish/genetics*
PubMed
24257628 Full text @ Development
Abstract

Gene modifications in animal models have been greatly facilitated through the application of targeted genome editing tools. The prokaryotic CRISPR/Cas9 type II genome editing system has recently been applied in cell lines and vertebrates. However, we still have very limited information about the efficiency of mutagenesis, germline transmission rates and off-target effects in genomes of model organisms. We now demonstrate that CRISPR/Cas9 mutagenesis in zebrafish is highly efficient, reaching up to 86.0%, and is heritable. The efficiency of the CRISPR/Cas9 system further facilitated the targeted knock-in of a protein tag provided by a donor oligonucleotide with knock-in efficiencies of 3.5-15.6%. Mutation rates at potential off-target sites are only 1.1-2.5%, demonstrating the specificity of the CRISPR/Cas9 system. The ease and efficiency of the CRISPR/Cas9 system with limited off-target effects make it a powerful genome engineering tool for in vivo studies.

Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping