PUBLICATION

Rapid screening of glomerular slit diaphragm integrity in larval zebrafish

Authors
Hentschel, D.M., Mengel, M., Boehme, L., Liebsch, F., Albertin, C., Bonventre, J.V., Haller, H., and Schiffer, M.
ID
ZDB-PUB-070820-10
Date
2007
Source
American journal of physiology. Renal physiology   293(5): F1746-F1750 (Journal)
Registered Authors
Hentschel, Dirk
Keywords
podocyte, nephrotic syndrome, puromycin aminonucleoside, CD2-associated protein, podocin
MeSH Terms
  • Adaptor Proteins, Signal Transducing/genetics
  • Animals
  • Cytoskeletal Proteins/genetics
  • Dextrans/pharmacokinetics
  • Diagnostic Techniques, Urological*
  • Edema/chemically induced
  • Fluorescein-5-isothiocyanate/analogs & derivatives
  • Fluorescein-5-isothiocyanate/pharmacokinetics
  • Gene Deletion
  • Injections
  • Intracellular Signaling Peptides and Proteins/genetics
  • Kidney Diseases/diagnosis*
  • Kidney Diseases/genetics
  • Kidney Diseases/metabolism
  • Kidney Diseases/pathology
  • Kidney Glomerulus*/drug effects
  • Kidney Glomerulus*/metabolism
  • Kidney Glomerulus*/pathology
  • Larva/anatomy & histology
  • Macromolecular Substances/metabolism
  • Membrane Proteins/genetics
  • Microscopy, Electron
  • Podocytes/pathology
  • Puromycin Aminonucleoside/administration & dosage
  • Puromycin Aminonucleoside/poisoning
  • Time Factors
  • Zebrafish/growth & development*
PubMed
17699558 Full text @ Am. J. Physiol. Renal Physiol.
Abstract
Gene array type experiments have identified large numbers of genes thought to be important for the integrity of the glomerular slit diaphragm. Confirmation of individual proteins has been limited by the expenses and time involved in generating transgenic or knock-out mice for each candidate. Here we present a functional screening assay based on the clearance of a 70kDa fluorescent dextran in another vertebrate system that is rapid and low in cost. In the pronephric glomerulus of larval zebrafish we demonstrate quantifiable loss of slit diaphragm integrity in a zebrafish model of puromycin aminonucleoside (PA) toxicity. In addition, after knock-down of CD2AP and podocin, two in mammals well characterized genetic contributors to podocyte differentiation, we observe glomerular loss of serum macromolecules similar to that seen in mammalian kidneys with inborn mutations in these genes. Increased filtration of 70 kDa FITC-labeled dextran correlates with effacement of podocyte foot processes in ultrastructural analysis. These findings document the value of the zebrafish model in genomics and pharmacological screening applications. Key words: podocyte, nephrotic syndrome, puromycin aminonucleoside, CD2AP, podocin.
Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping