PUBLICATION

A Positive Regulatory Loop between foxi3a and foxi3b Is Essential for Specification and Differentiation of Zebrafish Epidermal Ionocytes

Authors
Hsiao, C.D., You, M.S., Guh, Y.J., Ma, M., Jiang, Y.J., and Hwang, P.P.
ID
ZDB-PUB-070330-32
Date
2007
Source
PLoS One   2(1): e302 (Journal)
Registered Authors
Guh, Ying-Jey, Hsiao, Chung-Der, Hwang, Pung Pung, Jiang, Yun-Jin, Ma, Ming
Keywords
Embryos, Cell differentiation, Zebrafish, Ectoderm, Notch signaling, Inhibitions, Gene expression, Epidermis
MeSH Terms
  • Acid-Base Equilibrium/genetics
  • Animals
  • Arginase/genetics
  • Arginase/metabolism
  • Cell Differentiation
  • Epidermis/cytology*
  • Epidermis/enzymology
  • In Situ Hybridization
  • Ions/metabolism
  • Kidney/physiology
  • Potassium/physiology
  • Proton-Translocating ATPases/metabolism
  • Sodium-Potassium-Exchanging ATPase/metabolism
  • Zebrafish/genetics*
  • Zebrafish/growth & development
  • Zebrafish/metabolism
PubMed
17375188 Full text @ PLoS One
Abstract
BACKGROUND: Epidermal ionocytes play essential roles in the transepithelial transportation of ions, water, and acid-base balance in fish embryos before their branchial counterparts are fully functional. However, the mechanism controlling epidermal ionocyte specification and differentiation remains unknown. METHODOLOGY/PRINCIPAL FINDINGS: In zebrafish, we demonstrated that Delta-Notch-mediated lateral inhibition plays a vital role in singling out epidermal ionocyte progenitors from epidermal stem cells. The entire epidermal ionocyte domain of genetic mutants and morphants, which failed to transmit the DeltaC-Notch1a/Notch3 signal from sending cells (epidermal ionocytes) to receiving cells (epidermal stem cells), differentiates into epidermal ionocytes. The low Notch activity in epidermal ionocyte progenitors is permissive for activating winged helix/forkhead box transcription factors of foxi3a and foxi3b. Through gain- and loss-of-function assays, we show that the foxi3a-foxi3b regulatory loop functions as a master regulator to mediate a dual role of specifying epidermal ionocyte progenitors as well as of subsequently promoting differentiation of Na(+),K(+)-ATPase-rich cells and H(+)-ATPase-rich cells in a concentration-dependent manner. CONCLUSIONS/SIGNIFICANCE: This study provides a framework to show the molecular mechanism controlling epidermal ionocyte specification and differentiation in a low vertebrate for the first time. We propose that the positive regulatory loop between foxi3a and foxi3b not only drives early ionocyte differentiation but also prevents the complete blockage of ionocyte differentiation when the master regulator of foxi3 function is unilaterally compromised.
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