PUBLICATION

Functional repression of islet-2 by disruption of complex with Ldb impairs peripheral axonal outgrowth in embryonic zebrafish

Authors
Segawa, H., Miyashita, T., Hirate, Y., Higashijima, S., Chino, N., Uyemura, K., Kikuchi, Y., and Okamoto, H.
ID
ZDB-PUB-010705-5
Date
2001
Source
Neuron   30(2): 423-436 (Journal)
Registered Authors
Higashijima, Shin-ichi, Hirate, Yoshikazu, Kikuchi, Yutaka, Miyashita, Toshio, Okamoto, Hitoshi, Segawa, Hiroshi
Keywords
none
MeSH Terms
  • Animals
  • Axons/physiology*
  • Binding Sites
  • Embryo, Nonmammalian/physiology
  • Genes, Reporter
  • Green Fluorescent Proteins
  • Homeodomain Proteins/chemistry
  • Homeodomain Proteins/genetics
  • Homeodomain Proteins/metabolism*
  • LIM-Homeodomain Proteins
  • Luminescent Proteins/analysis
  • Luminescent Proteins/genetics
  • Mosaicism
  • Motor Neurons/physiology*
  • Nerve Tissue Proteins/genetics
  • Nerve Tissue Proteins/metabolism*
  • Neurons, Afferent/physiology*
  • Oligodeoxyribonucleotides, Antisense/pharmacology
  • Protein Biosynthesis
  • RNA, Messenger/genetics
  • Transcription Factors
  • Transcription, Genetic
  • Zebrafish/embryology*
  • Zebrafish/genetics*
PubMed
11395004 Full text @ Neuron
Abstract
Islet-2 is a LIM/homeodomain-type transcription factor of the Islet-1 family expressed in embryonic zebrafish. Two Islet-2 molecules bind to the LIM domain binding protein (Ldb) dimers. Overexpression of the LIM domains of Islet-2 or the LIM-interacting domain of Ldb proteins prevented binding of Islet-2 to Ldb proteins in vitro and caused similar in vivo defects in positioning, peripheral axonal outgrowth, and neurotransmitter expression by the Islet-2-positive primary sensory and motor neurons as the defects induced by injection of Islet-2-specific antisense morpholino oligonucleotide. These and other experiments, i.e., mosaic analysis, coexpression of full-length Islet-2, and overexpression of the chimeric LIM domains derived from two different Islet-1 family members, demonstrated that Islet-2 regulates neuronal differentiation by forming a complex with Ldb dimers and possibly with some other Islet-2-specific cofactors.
Genes / Markers
Figures
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Expression
Phenotype
Mutations / Transgenics
Human Disease / Model
Sequence Targeting Reagents
Fish
Antibodies
Orthology
Engineered Foreign Genes
Mapping