Morpholino

MO1-cxcl8b.1

ID
ZDB-MRPHLNO-140630-2
Name
MO1-cxcl8b.1
Previous Names
  • MO cxcl8-l2 E1/I1 (1)
Target
Sequence
5' - GCCAATGAGGGTAAGCAGATACTAA - 3'
Disclaimer
Although ZFIN verifies reagent sequence data, we recommend that you conduct independent sequence analysis before ordering any reagent.
Note
None
Genome Resources
None
Target Location
Genomic Features
No data available
Expression
Gene expression in Wild Types + MO1-cxcl8b.1
No data available
Phenotype
Phenotype resulting from MO1-cxcl8b.1
Phenotype of all Fish created by or utilizing MO1-cxcl8b.1
Phenotype Fish Conditions Figures
caudal fin Ab1-cxcl8b.1 labeling decreased amount, abnormal WT + MO1-cxcl8b.1 amputation: caudal fin Fig. S2 from de Oliveira et al., 2015
whole organism decreased life span, exacerbated WT + MO1-cxcl8b.1 bacterial treatment by injection: Salmonella enterica subsp. enterica serovar Typhimurium Fig. 1 from de Oliveira et al., 2015
neutrophil chemotaxis decreased occurrence, abnormal WT + MO1-cxcl8b.1 amputation: caudal fin Fig. 1 from de Oliveira et al., 2015
neutrophil chemotaxis decreased occurrence, abnormal i114Tg + MO1-cxcl8b.1 amputation: caudal fin Fig. 1Fig. 3 from de Oliveira et al., 2015
leukocyte migration involved in inflammatory response process quality, abnormal i114Tg + MO1-cxcl8b.1 physical alteration: caudal fin Fig. 2Fig. 4Fig. 5Fig. 6 from de Oliveira et al., 2013
defense response to bacterium decreased occurrence, abnormal i114Tg + MO1-cxcl8b.1 bacterial treatment by injection: Salmonella enterica subsp. enterica serovar Typhimurium Fig. 2 from de Oliveira et al., 2015
neutrophil chemotaxis decreased occurrence, abnormal i114Tg + MO1-cxcl8b.1 bacterial treatment by injection: Salmonella enterica subsp. enterica serovar Typhimurium Fig. 2 from de Oliveira et al., 2015
neutrophil-mediated killing of bacterium decreased occurrence, abnormal i114Tg + MO1-cxcl8b.1 bacterial treatment by injection: Salmonella enterica subsp. enterica serovar Typhimurium Fig. 2 from de Oliveira et al., 2015
intersegmental vessel morphology, abnormal i114Tg; y1Tg + MO1-cxcl8b.1 standard conditions Fig. S1 with image from Zuñiga-Traslaviña et al., 2017
caudal fin neutrophil decreased amount, abnormal i114Tg; y1Tg + MO1-cxcl8b.1 transection: caudal fin musculature Fig. 2 with image from Zuñiga-Traslaviña et al., 2017
blood neutrophil decreased amount, abnormal i114Tg; y1Tg + MO1-cxcl8b.1 transection: caudal fin musculature Fig. 2 with image from Zuñiga-Traslaviña et al., 2017
neutrophil chemotaxis disrupted, abnormal i114Tg; y1Tg + MO1-cxcl8b.1 transection: caudal fin musculature Fig. 2 with image from Zuñiga-Traslaviña et al., 2017
receptor internalization increased occurrence, abnormal cu101Tg + MO1-cxcl8b.1 transection: ventral fin fold Fig. 2 with image from Coombs et al., 2019
neutrophil chemotaxis decreased occurrence, abnormal i114Tg + MO1-cxcl8b.1 + MO1-lyn amputation: caudal fin Fig. 3 from de Oliveira et al., 2015
Citations