ZFIN Image: Chen et al., 2022, Fig. 2

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Figure Caption

Fig. 2

Tgs1 overexpression ameliorates the wing expansion defects caused by neuronal knockdown of Smn in Drosophila. (A) Examples of flies showing wing expansion failure upon nsyb-GAL4-driven (neur>) Smn RNAi. (B) Frequencies of the defective wing expansion phenotype in flies co-expressing a UAS-Smn RNAi (Smn RNAi) construct and the indicated UAS constructs driven by nsyb-GAL4 (neur>). UAS dTgs1 encodes GFP-tagged dTgs1; UAS hTGS1 encodes GFP-tagged human TGS1; UAS CTR is a control construct expressing the unrelated Mst protein. ΔSmn/+ flies are heterozygous for a deficiency of the Smn locus. Error bars: ± SEM. Numbers within and next to bars are the animals tested. P-values: one-way ANOVA with Tukey's multiple comparisons test.

Acknowledgments

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