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Fig. S7
Stable loss of srsf2b or srsf9 did not lead to the previously observed morphant developmental defects. The injection of sMOsrsf2b or sMOsrsf9 in corresponding mutants recapitulated the MO-associated phenotypes. A, Injection of sMOsrsf2b in wt and srsf2b-/- led to the same developmental defects including brain oedemas. B, Knock-down of srsf9 in wt and mutants using MOs generated vascular defects and the appearance of oedemas on the body. srsf2b-/- and srsf9-/- mutants were identified among offspring from heterozygous carriers by genotyping.
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Full text @ Nucleic Acids Res.