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Fig. S2 The ventral hindbrain is sensitive to low concentrations of RA. RNA in situ hybridizations showing vhnf1 (blue), and krox20 and pax2a (both in red) expression at the 3-somite stage (11 hpf). In embryos treated with 10 nM RA, which causes very mild, if any, patterning defects, the ventral-most hindbrain is specifically posteriorized, corresponding with the absence of cyp26c1 expression there (arrow in B). Scale bar: 100 μm.
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