Morpholino

MO1-scn5lab

ID
ZDB-MRPHLNO-100616-3
Name
MO1-scn5lab
Previous Names
  • AB-MO2 (1)
  • MO1-scn12ab
Target
Sequence
5' - AGTTTGTGCTGACCGGTGGTCTGGG - 3'
ZFIN BLAST NCBI BLAST Ensembl UCSC BLAT
Disclaimer
Although ZFIN verifies reagent sequence data, we recommend that you conduct independent sequence analysis before ordering any reagent.
Note
Splice-blocking MO.
Genome Resources
None
Target Location
Genomic Features
No data available
Expression
Gene expression in Wild Types + MO1-scn5lab
Expressed Gene Anatomy Figures
gata4 Fig. 4 from Chopra et al., 2010
hand2 Fig. 4 from Chopra et al., 2010
myh7 Fig. 5 from Chopra et al., 2010
myl7 Fig. 5 from Chopra et al., 2010
nkx2.5 Fig. 4,  Fig. 5 from Chopra et al., 2010
Phenotype
Phenotype resulting from MO1-scn5lab
Phenotype Fish Figures
atrium morphology, abnormal f1Tg + MO1-scn5lab Fig. 2 from Chopra et al., 2010
atrium orientation cardiac ventricle, abnormal WT + MO1-scn5lab + MO4-tp53 Fig. 2,  Fig. S6 from Chopra et al., 2010
blood circulation disrupted, abnormal f1Tg + MO1-scn5lab Fig. 2 from Chopra et al., 2010
cardiac muscle cell differentiation disrupted, abnormal WT + MO1-scn5lab Fig. 5 from Chopra et al., 2010
cardiac muscle cell proliferation decreased process quality, abnormal f2Tg + MO1-scn5lab + MO4-tp53 Fig. 6 from Bennett et al., 2013
cardiac ventricle morphology, abnormal f1Tg + MO1-scn5lab Fig. 2 from Chopra et al., 2010
eye apoptotic, abnormal WT + MO1-scn5lab Fig. S7 from Chopra et al., 2010
forebrain apoptotic, abnormal WT + MO1-scn5lab Fig. S7 from Chopra et al., 2010
head decreased size, abnormal WT + MO1-scn5lab Fig. 2,  Fig. 5,  Fig. S6 from Chopra et al., 2010
heart decreased size, abnormal WT + MO1-scn5lab + MO4-tp53 Fig. 2 from Bennett et al., 2013
heart has fewer parts of type cardiac muscle cell, abnormal f2Tg + MO1-scn5lab + MO4-tp53 Fig. 1 from Bennett et al., 2013
Fig. 3 from Chopra et al., 2010
heart hypoplastic, abnormal WT + MO1-scn5lab Fig. S7 from Chopra et al., 2010
heart malformed, abnormal WT + MO1-scn5lab Fig. S7 from Chopra et al., 2010
heart contraction decreased rate, abnormal f1Tg + MO1-scn5lab Fig. 2 from Chopra et al., 2010
heart looping arrested, abnormal f1Tg + MO1-scn5lab Fig. 2,  Fig. S6 from Chopra et al., 2010
heart looping decreased process quality, abnormal f2Tg + MO1-scn5lab + MO4-tp53 Fig. 1,  Fig. 2 from Bennett et al., 2013
heart primordium decreased size, abnormal WT + MO1-scn5lab Fig. 5 from Chopra et al., 2010
heart primordium has fewer parts of type cardiac muscle cell, abnormal WT + MO1-scn5lab Fig. 5 from Chopra et al., 2010
Phenotype of all Fish created by or utilizing MO1-scn5lab
Phenotype Fish Conditions Figures
cardiac muscle cell differentiation disrupted, abnormal WT + MO1-scn5lab standard conditions Fig. 5 from Chopra et al., 2010
heart primordium has fewer parts of type cardiac muscle cell, abnormal WT + MO1-scn5lab standard conditions Fig. 5 from Chopra et al., 2010
head decreased size, abnormal WT + MO1-scn5lab standard conditions Fig. 2,  Fig. 5,  Fig. S6 from Chopra et al., 2010
eye apoptotic, abnormal WT + MO1-scn5lab standard conditions Fig. S7 from Chopra et al., 2010
heart primordium decreased size, abnormal WT + MO1-scn5lab standard conditions Fig. 5 from Chopra et al., 2010
forebrain apoptotic, abnormal WT + MO1-scn5lab standard conditions Fig. S7 from Chopra et al., 2010
heart malformed, abnormal WT + MO1-scn5lab standard conditions Fig. S7 from Chopra et al., 2010
heart looping arrested, abnormal WT + MO1-scn5lab standard conditions Fig. S6 from Chopra et al., 2010
atrium orientation cardiac ventricle, abnormal WT + MO1-scn5lab standard conditions Fig. S6 from Chopra et al., 2010
heart hypoplastic, abnormal WT + MO1-scn5lab standard conditions Fig. S7 from Chopra et al., 2010
heart decreased size, abnormal WT + MO1-scn5lab + MO4-tp53 standard conditions Fig. 2 from Bennett et al., 2013
heart looping arrested, abnormal WT + MO1-scn5lab + MO4-tp53 standard conditions Fig. S6 from Chopra et al., 2010
atrium orientation cardiac ventricle, abnormal WT + MO1-scn5lab + MO4-tp53 standard conditions Fig. S6 from Chopra et al., 2010
heart looping decreased process quality, abnormal WT + MO1-scn5lab + MO4-tp53 standard conditions Fig. 2 from Bennett et al., 2013
atrium morphology, abnormal f1Tg + MO1-scn5lab standard conditions Fig. 2 from Chopra et al., 2010
heart contraction decreased rate, abnormal f1Tg + MO1-scn5lab standard conditions Fig. 2 from Chopra et al., 2010
cardiac ventricle morphology, abnormal f1Tg + MO1-scn5lab standard conditions Fig. 2 from Chopra et al., 2010
heart looping arrested, abnormal f1Tg + MO1-scn5lab standard conditions Fig. 2 from Chopra et al., 2010
atrium orientation cardiac ventricle, abnormal f1Tg + MO1-scn5lab standard conditions Fig. 2 from Chopra et al., 2010
blood circulation disrupted, abnormal f1Tg + MO1-scn5lab standard conditions Fig. 2 from Chopra et al., 2010
heart has fewer parts of type cardiac muscle cell, abnormal f2Tg + MO1-scn5lab standard conditions Fig. 3 from Chopra et al., 2010
cardiac muscle cell proliferation decreased process quality, abnormal f2Tg + MO1-scn5lab + MO4-tp53 standard conditions Fig. 6 from Bennett et al., 2013
heart looping decreased process quality, abnormal f2Tg + MO1-scn5lab + MO4-tp53 standard conditions Fig. 1 from Bennett et al., 2013
heart has fewer parts of type cardiac muscle cell, abnormal f2Tg + MO1-scn5lab + MO4-tp53 standard conditions Fig. 1 from Bennett et al., 2013
cardiac muscle cell proliferation decreased process quality, abnormal WT + MO1-scn5lab + MO1-tnnt2a + MO4-tp53 standard conditions Fig. 8 from Bennett et al., 2013
heart edematous, abnormal WT + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 4 from Huttner et al., 2013
heart looping decreased process quality, abnormal WT + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 4 from Huttner et al., 2013
heart contraction decreased process quality, abnormal WT + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 4 from Huttner et al., 2013
regulation of heart contraction disrupted, abnormal WT + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 4 from Chetaille et al., 2014
head decreased size, abnormal WT + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 2 from Chopra et al., 2010
heart contraction decreased rate, abnormal WT + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 4 from Chetaille et al., 2014
heart looping arrested, abnormal f1Tg + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 2 from Chopra et al., 2010
atrium orientation cardiac ventricle, abnormal f1Tg + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 2 from Chopra et al., 2010
blood circulation disrupted, abnormal f1Tg + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 2 from Chopra et al., 2010
atrium morphology, abnormal f1Tg + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 2 from Chopra et al., 2010
heart contraction decreased rate, abnormal f1Tg + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 2 from Chopra et al., 2010
cardiac ventricle morphology, abnormal f1Tg + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 2 from Chopra et al., 2010
heart has fewer parts of type cardiac muscle cell, abnormal f2Tg + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 3 from Chopra et al., 2010
heart looping decreased process quality, abnormal vcc1Tg + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 4 from Huttner et al., 2013
heart contraction decreased process quality, abnormal vcc1Tg + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 4 from Huttner et al., 2013
heart edematous, abnormal vcc1Tg + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 4 from Huttner et al., 2013
heart looping decreased process quality, abnormal vcc2Tg + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 4 from Huttner et al., 2013
heart edematous, abnormal vcc2Tg + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 4 from Huttner et al., 2013
heart contraction decreased process quality, abnormal vcc2Tg + MO1-scn12aa + MO1-scn5lab standard conditions Fig. 4 from Huttner et al., 2013
Citations
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